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Given AKT1s established role in insulin signaling, mitochondrial function, and energy metabolism, its upregulation may explain key pathological features of lipedema, including impaired oxidative phosphorylation, chronic inflammation, and altered adipocyte metabolism
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Perhaps this is due to the fact that autoantibodies are not a key link in the pathogenesis of HT, they begin to be produced already in response to damage to thyrocytes and are the result of an autoimmune response
However, none of the case reports included long-term follow-up, making it difficult to assess any delayed complications
